Explain why the underproduction of albumin by a cirrhotic liver contributes to excessive filtration, which leads to ascites. Refer again to the forces that determine net filtration pressure in the hepatic capillaries, and to the function of albumins.
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Explain why the underproduction of albumin by a cirrhotic liver contributes to excessive filtration, which leads to ascites. Refer again to the forces that determine net filtration pressure in the hepatic capillaries, and to the function of albumins.
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- Explain the steps of renin conversion to angiotensin II that take place once renin is secreted into the blood. Be specific and don’t use abbreviationsExplain what effect on total protein concentration you would expect to see during dehydration. Explain whether you would expect to see an increase or decrease in total protein concentration in haemorrhage.Explain why the colloid osmotic pressure of the plasma is much higher than the colloid osmotic pressure of interstitial fluid and why this is important in ensuring excess tissue fluid does not accumulate to cause edema.
- What would be the result of erythropoietin overproduction by the kidneys? Where are the kidneys located in the body? What is the medial indention on the kidneys called?Give written answer with explanation and conclusion A 65-year-old man with a 30-year history of alcoholism and liver disease visits his physician because of swelling in his abdomen. Which of the following sets of changes are most likely in the splanchnic circulation of this man?A) High capillary hydrostatic pressure; low plasma colloid osmotic pressureB) Low capillary hydrostatic pressure; high plasma colloid osmotic pressureC) High capillary hydrostatic pressure; high plasma colloid osmotic pressureD) Low capillary hydrostatic pressure; low plasma colloid osmotic pressureE) Normal capillary hydrostatic pressure; high plasma colloid osmotic pressureF) High capillary hydrostatic pressure; normal plasma colloid osmotic pressureWhy do people with hypertension keep a low-salt (low- sodium) diet? Explain further.
- With toxic damage to the liver cells, with a violation of its functions, the patient developed edema. What changes in the composition of blood plasma are the leading cause of the development of edema? Justify the answer.Explain the difference in the initiating event between the intrinsic pathway and the extrinsic pathway for blood coagulation.Would you expect to see an increase or decrease in total protein concentration in haemorrhage? Explain your reasoning.
- Give three features of the PCT necessary for its role in selective reabsorption. Explain the role fearures.(b) Explain why the colloid osmotic pressure of the plasma is much higher than the colloid osmotic pressure of interstitial fluid and why this is important in ensuring excess tissue fluid does not accumulate to cause edema.Illustrate the process of bilirubin formation starting from red blood cell destruction. (Indicate where Unconjugated bilirubin and Conjugated bilirubin is formed. Draw a line separating pre-renal, renal and post-renal.)