One mechanism by which anti-inflammatory medications like steroids work is by decreasing mast cell survival. Given what we learned about the inflammatory process, explain in detail how you think this type of steroid would work to block the process of inflammation
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One mechanism by which anti-inflammatory medications like steroids work is by decreasing mast cell survival.
Given what we learned about the inflammatory process, explain in detail how you think this type of steroid would work to block the process of inflammation
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- Explain why each choice (a-d) is correct or incorrect. All but one of the following occurs during the inflammatory response. Select the example below that does NOT describe the process of inflammation. a. Inflammation increases capillary permeability. b. Chemotaxis draws leucocytes to the site of injury. c. Vasoconstriction prevents excessive blood loss due to injury. d. Release of prostaglandins results in pain.Many of the inflammatory mediators produced by tissue macrophages at sites of infection act on the endothelial cells lining the blood vessel walls. An exception to this is (are) the: Cytokines that induce increased vascular permeability Chemokines that induce directed migration of blood monocytes Cytokines that induce increased expression of adhesion molecules TNF produced by tissue-resident sensor cells Bradykinin produced that causes painWhich of the following statements are TRUE about the process of inflammation? (Select all that apply) Inflammation can occur with only the innate immune response involved. Leukocytes or white blood cells are found everywhere in the body to carry out inflammation as needed Growth factor from non-skin cells is responsible for the formation of scar tissue in place of the damaged skin cells The increase of warmed blood to a site of injury comes from the hypothalamus Phagocytes engulf invading microbes and dead white blood cells Phagocytosis is used during inflammation to make sure there is no debris
- When surface barriers protecting the body are breached, the second line of defense (of the innate immune system) is activated. These internal defenses include inflammation and phagocytosis amongst other quick responses. Briefly explain acute inflammation in terms of vascular and cellular events. Use your understanding of blood vessels (smooth muscle and endothelial cells), chemical signalling and the interstitial space to help explain the four signs of inflammation - heat, redness, pain and swelling.During the chemotaxis phase of the inflammatory response,a. C-reactive protein is secreted by damaged parenchymal cells in the tissue, which attracts neutrophils, which secrete leukotrienes and prostaglandins to attract macrophages; a purulent exudate is formed.b. Band cells respond to hyperthermia by producing TNF-α and interleukins, which then cause a left shift that results in the accumulation of neutrophils within the tissue.c. Damaged parenchymal cells release prostaglandins and leukotrienes to attract macrophages, causing a left shift and the formation of a purulent exudate.d. Cytokines, such as TNF-α, are released by damaged parenchymal cells, while leukotrienes and prostaglandins produced by neutrophils cause smooth muscle constriction and the further accumulation of leukocytes.Mycobacteria are intracellular pathogens that have adapted to life inside phagocytic cells, such as macrophages. These intracellular bacteria are taken up by phagocytosis, similar to other pathogens, but the bacteria are not killed. One possible mechanism that could account for this immune evasion by mycobacteria is their ability to: Prevent induction of nitric oxide production in the phagosome Prevent the acidification of phagosomes Prevent the expression of antimicrobial peptides in the phagosome Prevent fusion of phagosomes with lysosomes Kill the macrophage before it kills them
- Cytokines and chemokines are major inflammatory mediators. Using examples explain their principal sources and effects.Describe the stages which lead to the extravasation of leukocytes during inflammation. In your description include the role of different adhesion molecules during the stages.In a specific type of tumor, cytotoxic T cells can recognize but not kill the tumor cell. Interaction of PD-L1 in tumor cells with PD1 in cytotoxic T cells plays a role in shaping the immune response by initiating signal transduction cascades in both cell types. What will happen if the signal transduction cascade in cytotoxic T cells is blocked? T cells will be activated and kill tumor cells T cells can recognize the cancer cells but will not be activated Tumor cell will undergo programmed cell death
- TH2 cells coordinate type 2 responses to expel intestinal helminths and repair tissue injury. The immune response to helminthic worm infections in the gastrointestinal tract requires specialized mechanisms due to the fact that helminths are too large to be ingested and destroyed by phagocytes. For example, cytokines made by TH2 cells elicit responses from multiple non-hematopoietic cell types that aid in parasite expulsion. Name two of these cell types and for each of them, their response to TH2-produced cytokines.Which of the following is NOT a true statement about inflammation? Substances released by invading bacteria themselves are the molecules that initiate inflammation The last stage in inflammation is tissue repair Substances released from damaged body cells attract phagocytic leukocytes to the area Inflammation can be triggered by physical injury or infection Phagocytic leukocytes migrate through the walls of blood vessels into surrounding tissuesThe immune system can be split into two major categories, the innate immune system and the adaptive immune system. Compare and contrast these two systems and their response to a bacterial pathogen that entered the body through a cut in the skin. In your answer describe two ways in which the innate immune system protects against this pathogen, as well as two ways that the adaptive immune system protectS against this pathogen. Be sure to name any cells/organs/tissues/structures that may be involved in each of these processes in order to receive full marks. This can be done as a list in point form.