Find and describe the actions of a chemotherapeutic agent that directly targets the cell cycle, specifically when during the cell cycle it works and what it interferes with.
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Find and describe the actions of a chemotherapeutic agent that directly targets the cell cycle, specifically when during the cell cycle it works and what it interferes with.
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- Which is true for cancer cells: 1) Cell death occurs after a determined number of cell divisions 2) Contact with other cells reduces chance of cell division 3) Cell division occurs in the presence of stop signals.name the enzymes of matrix biosynthesis, which should correct violations of the DNA structure in the norm, indicate its localization, connection with the phases of the cell cycle, energy sourcesThe cell cycle control system is a cycling set of molecules in the cell that triggers and coordinates key events in the cell cycle. This is all controlled by growth factors. a) Identify the major checkpoints in a cell and its function. b)Explain the role of the growth factor.
- Describe the three most important cell cycle checkpoints. Explain in a detailed manner and provide some sample scenarios.Please give information and explain the modeling of cancer with IPS cells (induced pluripotent stem cells). provide information about the processChoose all of the TRUE statements. Hint: 4 statements are true. Cytokinesis occurs the same way in both plant and animal cells. Sister chromatids are genetically identical. The term chromosome can be used to describe both chromosomes composed of one and two chromatids. Mitosis results in daughter cells that are genetically identical to the parent cell. All eukaryotic somatic cells are diploid. Cyclin dependent kinases are present but not active in cell cycle regulation without the presence of cyclin proteins. Chromosomes are always visible in the cell.
- Put a checkmark next to any of the following scenarios that would make it MORE likely that a cell would divide when it is not supposed to, increasing the chance that it would become cancerous. CHOOSE ALL THAT APPLY. A) A tumor suppressor gene is over-expressed B) A cyclin gene is mutated so that it is expressed at all times during the cell cycle C) A cyclin-dependent kinase gene is mutated so that in the Cdk is no longer dependent on its cyclin D) A Cdk gene is mutated so that the Cdk protein is not made E) Mutation of an oncogene that causes it to no longer be expressed F) A proto-oncogene gene is expressed at higher than normal levels G) The p53 gene is mutated so that no p53 protein is madeYou are studying a drug that leads to cell cycle arrest during mitosis. You examine levels of cyclin and cdk proteins in the cells treated with the drug, and untreated cells as control. Shown below are the Western blot results of protein expression. Drug No Drug Time (minutes) 15 30 45 60 75 0. 15 30 45 60 75 CDK Cydin Which of the following could be a mechanism by which the drug leads to cell cycle, arrest?If a drug interferes with the formation of microtubules , its effectiveness must be related to disruption of mitotic spindle formation suppression of cyclin production inhibition of DNA synthesis myosin denaturation and inhibition of cleavage formation
- Explain the cell division limit and its fail-safe functionWhich of the following statements best describes the process of phase separation in cells? Proteins can assemble as liquid droplets that are not bounded by a membrane in either the cytoplasm or nucleus A protein can separate into different organelles dependent on the phase of the cell cycle Proteins that are assembled in one phase of the cell cycle and degraded in the next are said to undergo phase separation Signal sequences can target proteins assembled in the cytoplasm into the lumen of the endoplasmic reticulumDiscuss the complete cell cycle in a human cell, mitosis and meiosis, and the regulatory components (i.e. the proteins associated with cellular checkpoints) of the cell cycle. Tumor growth results when the cell cycle checkpoints are ignored. Give an example of how tumor growth could result from either a loss-of-function or a gain-of-function mutation.