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what are the differnce between T1 and PD in MRI imaging?
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- Enumerate at least (any) 3 facts/indications about/for each of the following neuroimaging modalities: CT brain, MRI T1, MRI T2, MRI DWI, and MRI FLAIR.Please ASAP. Thanku. Enumerate at least (any) 3 facts/indications about/for each of the following neuroimaging modalities: CT brain, MRI T1, MRI T2, MRI DWI, and MRI FLAIR.Why it is difficult to determine the accuracy of a “memory” that is recovered in therapy?
- Discuss the concept of thought-controlled input devices and their potential in medical applications and assistive technology. What ethical considerations arise when using thought-controlled devices?A 55 year old man who is overweight, a heavy drinker and habitual smoker is brought in to the emergency department after he appeared confused and was slurring his speech at his workplace. Given the patient's lifestyle, the doctors suspect an ischemic stroke and immediately arrange for a scan to aid in the diagnosis. Discuss which imaging modality the doctor might use to establish if the patient had suffered a stroke and give reasons for your choice. [8] Describe the physical principles behind the imaging technique you have chosen. Explain the analytical process that could be used to confirm whether the stroke was ischemic rather than hemorrhagic. As a further precaution and to inform the management of the patient's condition going forward, the doctor wants to assess whether the patient has atherosclerosis in his carotid artery. Describe the basis of an imaging technique that would be used to give this additional information.Describe how lesioning studies and brain stimulation are used to study the brain.
- Could you please mention/explain some limitations and extensions (three each) associated with the following claim and research question. Claim: Gene therapy can reverse ageing. Research question: How does the injection of BDNF in the brain through gene therapy help treat Alzheimer's disease in people over the age of 65? please asapExplain what is neuroplasticity and why is this important in the case of Phineas Gage?Link: https://doi.org/10.1186/s12868-022-00692-1 A. In the effect of mitoxantrone on histopathological changes in the brain section of the results section the authors wrote “In Fig. 4, the demyelination of (A-C) the cerebellum and (F-H) brain stem was evaluated using LFB staining and median scored 2.5 in both sedentary and exercised untreated EAE rats indicating extensive demyelination.” Why was demyelination evaluated in this study? B. In the effect of mitoxantrone on histopathological changes in the brain section of the results section the authors wrote “Post-administration of mitoxantrone to sedentary and exercised groups smaller patches of demyelination with microcyst formation and lymphocytic infiltrates were seen; and bare unmyelinated axons were fewer (Figs. 3D, E, I, J and 4D, E, I, J).” Based on this quote, what is mitoxantrone doing exactly/directly?