Why was Mendel’s success dependent on his studying characteristics that exhibit only two easily distinguished phenotypes, such as white versus gray seed coats and round versus wrinkled seeds? Would he have been less successful if he had instead studied traits such as seed weight or leaf length, which vary much more in their phenotypes? Explain your answer.
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Why was Mendel’s success dependent on his studying characteristics that exhibit only two easily distinguished
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- Why was Mendel’s success dependent on his studying characteristics that exhibit only two easily distinguished phenotypes, such as white versus gray seed coats and round versus wrinkled seeds? Would he have been less successful if he had instead studied traits such as seed weight or leaf length, which vary much more in their phenotype?Why was Mendel’s success dependent on his studying characteristics that exhibit only two easily distinguished phenotypes, such as white versus gray seed coats and round versus wrinkled seeds? Would he have been less successful if he had instead studied traits like seed weight or length of the leaves, which vary much more in their phenotypes? Explain your answer.Using Mendel’s lines of peas, describe how you would use a test cross experimental design to determine the genotype of a pea plant with purple flowers. Be very specific in describing your experimental design and how you would interpret your results based on the proportion of offspring phenotypes. Make sure to include all possible offspring phenotypes in your answer and what the proportion of phenotypes tells you about the purple plant genotype.
- Mendel describes subjecting each of the 34 varieties of peas he obtained to a two-year trial. During this time he let the plants self-fertilize and observed their offspring. What was he looking for, and what was the purpose of doing this two-year trial? Explain what Mendel means when he writes that the 3:1 ratio observed in the first generation from the hybrids "resolves itself" into a ratio of 2:1:1Use the following information to answer the next question. A Venn Diagram Showing the Relationship Between Oogenesis and Spermatogeneis. Oogenesis 1 NOTE: Similarities are represented by area 2. Differences are represented by areas 1 or 3. Match the numbered regions with the descriptions below. Spermatogenesis 3 Four viable gametes produced: Occurs in gonads: Daughter cells equal in size Unequal cytoplasmic division ▶In Mendel's genetic experiments many characteristics of the plants were quantified, such as their height (tall or short), flower position (axial or terminal), and seed color (colored or white). Suppose 100 plants are measured, and 37 are tall with axial flowers and colored seeds. Further, suppose a total of 26 plants have white seeds, 4 of them being short and 7 having terminal flowers. There are no short plants with terminal flowers, and the total number of short plants is 12. What is the probability a randomly selected plant will have flowers in the terminal position? Round your answer to THREE DECIMAL places. Also, double check your answer before posting. thank you
- As you know Mendel’s main contribution to genetics was his proposing a model of particulate inheritance. This discovery contradicted the widely held belief that blending inheritance was the true theory that explained hereditary traits. One advantage that Mendel had in choosing garden peas (Pisum sativum) was that he could either allow the pea flowers on a plant to self-pollinate or he could deliberately cross-pollinate the flowers. For his single locus crosses of pure-breeding lines, he would take the F2 offspring of the dominant phenotype (such as yellow seeds) and ensure that each yellow-seeded plant would self-pollinate. He was able to show that 1/3 of all the yellow-seeded plants in this generation bred true while the other 2/3 of the yellow seeded plants showed segregation. Do you believe that this extra experiment gave additional important evidence for the particulate theory of inheritance or did the offspring from the F1 x F1 cross provide enough evidence of Mendel’s First Law?…Mendel began his work with pairs of varieties from the breeders that differed from each other in just one trait (corresponding to one gene difference, as we now know) out of the 7 traits he studied. These pairs could be obtained by self-pollinating an individual from an advanced generation that was pure-breeding for 6 of the traits but heterozygous for the 7th. i. What is the probability of finding an example plant in the F10 generation? ii. What is the probability of finding an example plant in the F4 generation? [Note: this probability is higher than that for the F10 generation or the F3 generation. What qualitative reason might there be?In this program, you are provided with phenotype pair counts of F2 offspring at two research institutes. The key different between this work and previous work is that now we consider two genes instead of one. The phenotype pairs are the (shape, color) of peas from a pea plant. It turns out that there are two separate genes that code for these phenotypes. We shall call them Shape and Color. Gregor Mendel originally recorded these experiments in green peas. Using the notation: R = Round (dominant) allele at Shape gene; r = Wrinkled (recessive) allele at Shape gene; Y = Yellow (dominant) allele at Color gene; y = Green (recessive) allele at Color gene; then the shape and color of any pea can be determined by studying the genotypes at each gene. It turns out that, when one mates a plant that is homozygous for the dominant alleles (RRYY) with a plant that is homozygous for the recessive alleles (rryy), the F1 generation are heterozygous at both genes, as with a single gene disorder.…
- Pedigree analysis is a fundamental tool for investigating whether or not a trait is following a Mendelian pattern of inheritance. It can also be used to help identify individuals within a family who may be at risk for the trait. Adam and Sarah, a young couple of Eastern European Jewish ancestry, went to a genetic counselor because they were planning a family and wanted to know what their chances were for having a child with a genetic condition. The genetic counselor took a detailed family history from both of them and discovered several traits in their respective families. Sarahs maternal family history is suggestive of an autosomal dominant pattern of cancer predisposition to breast and ovarian cancer because of the young ages at which her mother and grandmother were diagnosed with their cancers. If a mutant allele that predisposed to breast and ovarian cancer was inherited in Sarahs family, she, her sister, and any of her own future children could be at risk for inheriting this mutation. The counselor told her that genetic testing is available that may help determine if this mutant allele is present in her family members. Adams paternal family history has a very strong pattern of early onset heart disease. An autosomal dominant condition known as familial hypercholesterolemia may be responsible for the large number of deaths from heart disease. As with hereditary breast and ovarian cancer, genetic testing is available to see if Adam carries the mutant allele. Testing will give the couple more information about the chances that their children could inherit this mutation. Adam had a first cousin who died from Tay-Sachs disease (TSD), a fatal autosomal recessive condition most commonly found in people of Eastern European Jewish descent. Because TSD is a recessively inherited disorder, both of his cousins parents must have been heterozygous carriers of the mutant allele. If that is the case, Adams father could be a carrier as well. If Adams father carries the mutant TSD allele, it is possible that Adam inherited this mutation. Because Sarah is also of Eastern European Jewish ancestry, she could also be a carrier of the gene, even though no one in her family has been affected with TSD. If Adam and Sarah are both carriers, each of their children would have a 25% chance of being afflicted with TSD. A simple blood test performed on both Sarah and Adam could determine whether they are carriers of this mutation. Would you decide to have a child if the test results said that you carry the mutation for breast and ovarian cancer? The heart disease mutation? The TSD mutation? The heart disease and the mutant alleles?Pedigree analysis is a fundamental tool for investigating whether or not a trait is following a Mendelian pattern of inheritance. It can also be used to help identify individuals within a family who may be at risk for the trait. Adam and Sarah, a young couple of Eastern European Jewish ancestry, went to a genetic counselor because they were planning a family and wanted to know what their chances were for having a child with a genetic condition. The genetic counselor took a detailed family history from both of them and discovered several traits in their respective families. Sarahs maternal family history is suggestive of an autosomal dominant pattern of cancer predisposition to breast and ovarian cancer because of the young ages at which her mother and grandmother were diagnosed with their cancers. If a mutant allele that predisposed to breast and ovarian cancer was inherited in Sarahs family, she, her sister, and any of her own future children could be at risk for inheriting this mutation. The counselor told her that genetic testing is available that may help determine if this mutant allele is present in her family members. Adams paternal family history has a very strong pattern of early onset heart disease. An autosomal dominant condition known as familial hypercholesterolemia may be responsible for the large number of deaths from heart disease. As with hereditary breast and ovarian cancer, genetic testing is available to see if Adam carries the mutant allele. Testing will give the couple more information about the chances that their children could inherit this mutation. Adam had a first cousin who died from Tay-Sachs disease (TSD), a fatal autosomal recessive condition most commonly found in people of Eastern European Jewish descent. Because TSD is a recessively inherited disorder, both of his cousins parents must have been heterozygous carriers of the mutant allele. If that is the case, Adams father could be a carrier as well. If Adams father carries the mutant TSD allele, it is possible that Adam inherited this mutation. Because Sarah is also of Eastern European Jewish ancestry, she could also be a carrier of the gene, even though no one in her family has been affected with TSD. If Adam and Sarah are both carriers, each of their children would have a 25% chance of being afflicted with TSD. A simple blood test performed on both Sarah and Adam could determine whether they are carriers of this mutation. Would you want to know the results of the cancer, heart disease, and TSD tests if you were Sarah and Adam? Is it their responsibility as potential parents to gather this type of information before they decide to have a child?Pedigree analysis is a fundamental tool for investigating whether or not a trait is following a Mendelian pattern of inheritance. It can also be used to help identify individuals within a family who may be at risk for the trait. Adam and Sarah, a young couple of Eastern European Jewish ancestry, went to a genetic counselor because they were planning a family and wanted to know what their chances were for having a child with a genetic condition. The genetic counselor took a detailed family history from both of them and discovered several traits in their respective families. Sarahs maternal family history is suggestive of an autosomal dominant pattern of cancer predisposition to breast and ovarian cancer because of the young ages at which her mother and grandmother were diagnosed with their cancers. If a mutant allele that predisposed to breast and ovarian cancer was inherited in Sarahs family, she, her sister, and any of her own future children could be at risk for inheriting this mutation. The counselor told her that genetic testing is available that may help determine if this mutant allele is present in her family members. Adams paternal family history has a very strong pattern of early onset heart disease. An autosomal dominant condition known as familial hypercholesterolemia may be responsible for the large number of deaths from heart disease. As with hereditary breast and ovarian cancer, genetic testing is available to see if Adam carries the mutant allele. Testing will give the couple more information about the chances that their children could inherit this mutation. Adam had a first cousin who died from Tay-Sachs disease (TSD), a fatal autosomal recessive condition most commonly found in people of Eastern European Jewish descent. Because TSD is a recessively inherited disorder, both of his cousins parents must have been heterozygous carriers of the mutant allele. If that is the case, Adams father could be a carrier as well. If Adams father carries the mutant TSD allele, it is possible that Adam inherited this mutation. Because Sarah is also of Eastern European Jewish ancestry, she could also be a carrier of the gene, even though no one in her family has been affected with TSD. If Adam and Sarah are both carriers, each of their children would have a 25% chance of being afflicted with TSD. A simple blood test performed on both Sarah and Adam could determine whether they are carriers of this mutation. If Sarah carries the mutant cancer allele and Adam carries the mutant heart disease allele, what is the chance that they would have a child who is free of both diseases? Are these good odds?