Blood sugar homeostasis: Indicate the order of events that will happen following blood glucose increase after a meal.Increased blood glucose1234LO56::::Glucose transport via GLUT4Glucose transport into pancreatic cellsInsulin binds to insulin receptorInsulin release from pancreatic beta-cellsSecond messenger signaling pathway is activatedGLUT4 moves to plasma membraneDecreased blood glucose AND muscle makes ATP and glycogen

After a meal, blood glucose increases. To maintain blood glucose level, insulin is released by the…

Answered: Blood sugar homeostasis: Indicate the…

Principles Of Pharmacology Med Assist

6th Edition

ISBN:9781337512442

Author:RICE

Publisher:RICE

Chapter21: Medications Used For Musculoskeletal System Disorders

Section: Chapter Questions

Problem 24RQ

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. Explain why insulin can stimulate the activation PKB of but not the activation of PK.
Which might be a physiological phenotype associated with loss of function of GLUT1? inability of glucose to enter the intestinal cells from the intestinal lumen and high glucose excreted accumulation of glucose in the intestinal epithelial cytoplasm loss of glucose transport into somatic tissues (such as neurons in the brain) low glucose levels in the bloodstream
Sequence: The Fate of Glucose Increased glycolysis in beta Glucose absorbed through intestines to bloodstream Plasma glucose level increases and glucose enters beta cells cell increases ATP production Salivary Amylase breaks down carbs into dextrin/maltose Beta cell membrane Increased beta cell ATP shuts K channel depolarization Plasma glucose levels decrease as it enters GLUT Beta cell voltage gated calcium channel opens Calcium enters into beta cell 4 cells Insulin releases into bloodstream Person Eats Glucose enters Carbohydrates GLUT4 cell Insulin binds to insulin GLUT 4 complex Pancreatic Amylase breaks down dextrin to glucose receptors moves to membrane étv
Would a metabolic rational for a child who has a similar diet to other children but after infections or vigorous exercise has low blood glucose levels and increased lactic acid in the blood along with potential lethargy, shakiness and irritability be type 1 diabetes? Noting that an effective treatment for them is alanine supplementation in the diet.
Define glucose-sparing effect
describe a ketogenic diet. what are the pros and cons of such a diet. how is ketosis related to diabetes mellitus?
Insulin signals activate all of the following enzymes except: Phosphoinositide 3-kinase (PI3K) Glycogen synthase kinase 3 (GSK3) Protein kinase B (PKB; or Akt) All of the above enzymes are activated by insulin signals Citrate move in and out of mitochondria via a specific transport protein. What would happen if aninhibitor completely inhibited this transport system? Fatty acid biosynthesis would slow down Ketone body synthesis would slow down Both A and B Neither A nor B
Endocrine & Signalling 26. HO- NH2 HO- The molecule above is a hormone. Select FOUR statements that are correct with regard to this moelcule below. This is the least active form of this hormone There is another more active form of this hormone This molecule is derived from tyrosine This hormone stimulates fat metabolism Biosynthesis of this molecule requires iodine This molecule is a tryptophan metabolite This hormone targets two specific receptors This hormone stimulates fat storage This molecule is derived from cholesterol This hormone is synthesised in the adrenals
Explain why insulin is required for adipocytes to synthesize triacylglycerols from fatty acids.
Compare the localization of GLUT4 with that of GLUT2 and GLUT3, and explain why these localizations are important in the response of muscle,adipose tissue, brain, and liver to insulin
Matching – Draw lines to connect the terms Glucose-6-phosphate Low ATP/AMP ratio Fructose-1,6-Bisphosphate Has a High Km for glucose High ATP/AMP ratio Glucokinase Inhibits hexokinase Stimulate pyruvate Kinase Inhibits PFK-1 Stimulates PFK-1
Explain why adipocytes need glucose as well as fatty acids in order to synthesize triacylglycerols.

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